One Wire Phenomenon
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- Oct 21, 2020
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I know I said I'm not going to post anymore but I couldn't help it with this one I'm sorry.
If this can be possible in nature it's possible in humans aswell as in biologically and spiritually. This plant has the characteristics of a parasitic Jew. Im pretty sure there are non human soul species out there that are the same in many ways.They latch onto a soul and later become a part of it like it's not even there like a wart or genetical disease or something.The scientific research has shown this plant parasite has evolved so much for so long that it's genome cannot be trace back to its original. I also think these parasites that are found in nature and in humans will unlock the secrets of hacking that would eventually lead to a bio-microchip for humans. Think about the greys and their space craft when they interact with it it becomes like a living extension of themselfs.
Here is the articles
"An international team of researchers has sequenced and analyzed the genome of Sapria himalayana, a rare holoparasitic flowering plant in the family Rafflesiaceae. Their findings, published in the journal Current Biology, offer a unique perspective into how flowering plant genomes can be altered to fit an extreme form of plant parasitism.
Sapria himalayana represents the extreme manifestation of the parasitic lifestyle, being totally dependent on its host for water nutrients and photosynthates.
Its flowers are about 20 cm (8 inches) across, bright red in color covered with sulfur-yellow spots. They appear above the ground and have a putrid odor.
Sapria himalayana’s more famous cousin, Rafflesia arnoldii, produces flowers up to 1 m (3.3 feet) in diameter, the largest in the world.
“In many ways, it’s a miracle that these plants exist today, let alone that they seem to have persisted for tens of millions of years,” said Professor Charles Davis, a researcher in the Department of Organismic and Evolutionary Biology at Harvard University and curator of vascular plants in the Harvard University Herbaria.
These findings bring unique perspectives into the number and kind of genes it takes to be an endoparasite, along with offering new insights into how far the genomes of flowering plants can be altered and still remain functional.
What struck the researchers immediately was the degree of gene loss Sapria himalayana experienced as they abandoned their bodies and adapted to become endoparasites.
About 44% of all genes found in most flowering plants are absent in the genome of Sapria himalayana. That extent of gene loss is more than four times the degree of loss in other plant parasites.
Many of the genes lost include what are considered the key genes responsible for photosynthesis, which converts light into energy.
At the same time, the data demonstrated an underlying evolutionary convergence to becoming a parasite because Sapria himalayana and the parasitic plants the scientists compared them to lost many of the same types of genes despite evolving separately.
They also identified dozens of genes that came into the Sapria himalayana genome through a process called horizontal (or lateral) gene transfer instead of the traditional parent-to-offspring transmission.
Basically, it means Sapria himalayana stole DNA from their host instead of getting it passed down to them.
The authors then reconstructed the lateral gene transfers they detected to put together a hidden history of former hosts going back millions of years.
“We concluded that there is a common genomic or genetic roadmap to how plant parasites evolve,” said Dr. Cai Liming, a researcher at the University of California, Riverside."
https://pubmed.ncbi.nlm.nih.gov/33485466/
New Study Shows How Dormant Toxoplasma Parasites Manipulate Neurons to Survive
Dec 27, 2021 by News Staff / Source
Toxoplasma gondii develops a latent infection in the muscle and central nervous system that acts as a reservoir for acute-stage reactivation in vulnerable patients. Until now little is understood about how these parasites manipulate host cells during latent infection and the impact this has on survival. New research led by the Walter and Eliza Hall Institute of Medical Research, the University of Melbourne and the Wellcome Center for Anti-Infectives Research shows that Toxoplasma gondii is able to lay dormant and undetected inside neurons and muscle cells by releasing proteins that switch off the cells’ ability to alert the immune system. This discovery could pave the way for new drug targets to treat patients suffering from chronic toxoplasmosis infections.
Microscopic cysts containing Toxoplasma gondii develop in the tissues of many vertebrates. Here, in mouse brain tissue, thousands of bradyzoites (stained red) are enveloped by a thin parasite cyst wall. Image credit: Jitinder P. Dubey.
Microscopic cysts containing Toxoplasma gondii develop in the tissues of many vertebrates. Here, in mouse brain tissue, thousands of bradyzoites (stained red) are enveloped by a thin parasite cyst wall. Image credit: Jitinder P. Dubey.
“Toxoplasma gondii parasites often lay dormant (called bradyzoites) in the brain of the host animal, where they could reactivate to cause severe disease,” said Ushma Ruparel, a Ph.D. student in the Walter and Eliza Hall Institute of Medical Research and the Department of Medical Biology at the University of Melbourne.
“We know that Toxoplasma gondii parasites make their own molecules and proteins that it exports into the host cell to protect itself from detection by the immune system.”
“While this is well-understood in the acute, initial stage of infection, how Toxoplasma gondii hijacks its host cells in its dormant form was unknown.”
With colleagues who have expertise in genomics and advanced imaging, Ruparel and colleagues showed that the parasite exports proteins called Inhibitor of STAT1 transcription (IST) to the host cell to suppress immune signals.
“IST plays a key role in limiting interferon signaling in bradyzoites, to protect host cells from immune-mediated cell death, which is essential to the body’s fight against Toxoplasma gondii,” said Dr. Chris Tonkin, also from the Walter and Eliza Hall Institute of Medical Research and the Department of Medical Biology at the University of Melbourne.
“Interferon is the beacon molecule of the immune system. It gives off a blinking sign to notify the immune system that the body has been infected with a range of diseases, including Toxoplasma gondii.”
“Interferon is crucial in the fight against disease. However, the cunning parasite tries to turn this signal off during the latent period, as this is the only way it can survive.”
“While the immune system is determined to rid the infection from the body, the parasite only has survival on its mind. So, it’s essentially a tug-of-war, a molecular battle.”
“Our next step is to figure out what it is about chronic infections that make those changes really unique to chronic infections,” he added.
“We have found one specific parasite protein that seems to have a role, but it doesn’t seem to account for all changes, and we want to explore if there are any others.”
If this can be possible in nature it's possible in humans aswell as in biologically and spiritually. This plant has the characteristics of a parasitic Jew. Im pretty sure there are non human soul species out there that are the same in many ways.They latch onto a soul and later become a part of it like it's not even there like a wart or genetical disease or something.The scientific research has shown this plant parasite has evolved so much for so long that it's genome cannot be trace back to its original. I also think these parasites that are found in nature and in humans will unlock the secrets of hacking that would eventually lead to a bio-microchip for humans. Think about the greys and their space craft when they interact with it it becomes like a living extension of themselfs.
Here is the articles
"An international team of researchers has sequenced and analyzed the genome of Sapria himalayana, a rare holoparasitic flowering plant in the family Rafflesiaceae. Their findings, published in the journal Current Biology, offer a unique perspective into how flowering plant genomes can be altered to fit an extreme form of plant parasitism.
Sapria himalayana represents the extreme manifestation of the parasitic lifestyle, being totally dependent on its host for water nutrients and photosynthates.
Its flowers are about 20 cm (8 inches) across, bright red in color covered with sulfur-yellow spots. They appear above the ground and have a putrid odor.
Sapria himalayana’s more famous cousin, Rafflesia arnoldii, produces flowers up to 1 m (3.3 feet) in diameter, the largest in the world.
“In many ways, it’s a miracle that these plants exist today, let alone that they seem to have persisted for tens of millions of years,” said Professor Charles Davis, a researcher in the Department of Organismic and Evolutionary Biology at Harvard University and curator of vascular plants in the Harvard University Herbaria.
These findings bring unique perspectives into the number and kind of genes it takes to be an endoparasite, along with offering new insights into how far the genomes of flowering plants can be altered and still remain functional.
What struck the researchers immediately was the degree of gene loss Sapria himalayana experienced as they abandoned their bodies and adapted to become endoparasites.
About 44% of all genes found in most flowering plants are absent in the genome of Sapria himalayana. That extent of gene loss is more than four times the degree of loss in other plant parasites.
Many of the genes lost include what are considered the key genes responsible for photosynthesis, which converts light into energy.
At the same time, the data demonstrated an underlying evolutionary convergence to becoming a parasite because Sapria himalayana and the parasitic plants the scientists compared them to lost many of the same types of genes despite evolving separately.
They also identified dozens of genes that came into the Sapria himalayana genome through a process called horizontal (or lateral) gene transfer instead of the traditional parent-to-offspring transmission.
Basically, it means Sapria himalayana stole DNA from their host instead of getting it passed down to them.
The authors then reconstructed the lateral gene transfers they detected to put together a hidden history of former hosts going back millions of years.
“We concluded that there is a common genomic or genetic roadmap to how plant parasites evolve,” said Dr. Cai Liming, a researcher at the University of California, Riverside."
https://pubmed.ncbi.nlm.nih.gov/33485466/
New Study Shows How Dormant Toxoplasma Parasites Manipulate Neurons to Survive
Dec 27, 2021 by News Staff / Source
Toxoplasma gondii develops a latent infection in the muscle and central nervous system that acts as a reservoir for acute-stage reactivation in vulnerable patients. Until now little is understood about how these parasites manipulate host cells during latent infection and the impact this has on survival. New research led by the Walter and Eliza Hall Institute of Medical Research, the University of Melbourne and the Wellcome Center for Anti-Infectives Research shows that Toxoplasma gondii is able to lay dormant and undetected inside neurons and muscle cells by releasing proteins that switch off the cells’ ability to alert the immune system. This discovery could pave the way for new drug targets to treat patients suffering from chronic toxoplasmosis infections.
Microscopic cysts containing Toxoplasma gondii develop in the tissues of many vertebrates. Here, in mouse brain tissue, thousands of bradyzoites (stained red) are enveloped by a thin parasite cyst wall. Image credit: Jitinder P. Dubey.
Microscopic cysts containing Toxoplasma gondii develop in the tissues of many vertebrates. Here, in mouse brain tissue, thousands of bradyzoites (stained red) are enveloped by a thin parasite cyst wall. Image credit: Jitinder P. Dubey.
“Toxoplasma gondii parasites often lay dormant (called bradyzoites) in the brain of the host animal, where they could reactivate to cause severe disease,” said Ushma Ruparel, a Ph.D. student in the Walter and Eliza Hall Institute of Medical Research and the Department of Medical Biology at the University of Melbourne.
“We know that Toxoplasma gondii parasites make their own molecules and proteins that it exports into the host cell to protect itself from detection by the immune system.”
“While this is well-understood in the acute, initial stage of infection, how Toxoplasma gondii hijacks its host cells in its dormant form was unknown.”
With colleagues who have expertise in genomics and advanced imaging, Ruparel and colleagues showed that the parasite exports proteins called Inhibitor of STAT1 transcription (IST) to the host cell to suppress immune signals.
“IST plays a key role in limiting interferon signaling in bradyzoites, to protect host cells from immune-mediated cell death, which is essential to the body’s fight against Toxoplasma gondii,” said Dr. Chris Tonkin, also from the Walter and Eliza Hall Institute of Medical Research and the Department of Medical Biology at the University of Melbourne.
“Interferon is the beacon molecule of the immune system. It gives off a blinking sign to notify the immune system that the body has been infected with a range of diseases, including Toxoplasma gondii.”
“Interferon is crucial in the fight against disease. However, the cunning parasite tries to turn this signal off during the latent period, as this is the only way it can survive.”
“While the immune system is determined to rid the infection from the body, the parasite only has survival on its mind. So, it’s essentially a tug-of-war, a molecular battle.”
“Our next step is to figure out what it is about chronic infections that make those changes really unique to chronic infections,” he added.
“We have found one specific parasite protein that seems to have a role, but it doesn’t seem to account for all changes, and we want to explore if there are any others.”
